1955 was when the first polio vaccine was introduced. It was Dr Salk’s “inactivated”polio vaccine. The Salk vaccine was approved just after being developed and fast-tracked into licensure by the US Department of Health, Education, and Welfare. This approval process took a record-breaking two hours. [1]
This approval process and the way it was put on the market was fascinating and well documented. For example, “Jonas Salk had a paper in which he argued that all the virus was inactivated and that there was no live virus left. But the sixth lot was not listed. And so I said that something was wrong. He cut out data in order not to show what happened to some lots…Well, NFIP did form an advisory committee. And they reformed it five or six times. Each time somebody didn’t agree, they dropped them and got somebody who might agree. By the time they were done forming the committee, everybody on it was distinguished, but very agreeable.” [2]
As a result of ignoring the warnings by highly qualified scientists who repeatedly and publicly explained why and how the inactivation process was flawed from the beginning, the vaccine infected, paralyzed, and killed children and their household contacts. This became known as the “Cutter Incident”.
Between April 17 and June 30, 1955, 260 poliomyelitis cases were documented after inoculation of about 400,000 persons with the Cutter vaccine. 94 cases were among vaccinees, 126 among family contacts, and 40 among community contacts. An estimate of the case-infection ratio is in the range of 1 case per 100 to 600 injected infections. [3]
“In the end, at least 220,000 people were infected with live polio virus contained in Cutter’s vaccine; 70,000 developed muscle weakness, 164 were severely paralyzed, 10 were killed. Seventy five percent of Cutter’s victims were paralyzed for the rest of their lives.” It is admitted by the most staunch vaccine advocates that, “…the disease caused by Cutter’s vaccine was worse than the disease caused by the natural polio virus.” [4]
This is a conservative estimate of the damage. Scientists at the time admitted this vaccine may have caused much more damage than was being reported.
Most mainstream stories of polio ignore the Cutter Incident altogether, but if they do cover it, they blame Cutter laboratories. They ignore the fact that all manufacturers had documented difficulty killing the virus in their vaccines, before and after the disaster. [5] And Cutter was not the only lab producing an “inactivated” vaccine that was injected into children and caused paralysis. [6]
The inactivated polio vaccine is still inactivated by the use of formaldehyde. The balance must be difficult to achieve. You must kill enough of the virus that it doesn’t cause paralysis but leave enough so that an antibody response is obtained and the more formaldehyde you use, the more toxic the vaccine becomes. Formaldehyde is used in embalming, is listed as a carcinogen, and is toxic to life which is why it is useful in “killing” viruses.
Children in the US receive 4 doses of inactivated polio vaccine by the time they are 5 (3 of those by 15 months). [7] The safety monitoring of the vaccine in clinical trials given today was a short, two-day process. [8]
The oral polio vaccine (OPV) was first introduced in 1961 by Dr. Albert Sabin. It replaced the inactivated vaccines as the more popular option for the next few decades and is attributed the success of disease eradication here in the US. [9]
It is said that for 1 in 750,000 cases, the live virus will mutate back into a virulent form and cause paralysis in its victim. This is called vaccine associated paralytic polio (VAPP) and it looks like this:
“Patient 1 was a 17-month-old girl. She had exhibited antibody deficiency and thus received inactivated polio vaccine (IPV). She was a household contact of a healthy OPV-vaccinated sibling. Limited data indicated that paralysis became evident in June 1995. All 3 fecal specimens collected 3–6 days after onset of paralysis yielded VDPV type 2. Patient died 8 days after onset of paralysis.
“Patient 2 was a boy born in January 2005. He received 4 doses of OPV, administered at birth and at 2, 4, and 6 months of age. In August 2005, he was hospitalized with irritability, drowsiness, hypotonia, and right paraparesis. Two collected fecal specimens tested were positive for VDPV type 2. His condition deteriorated during the next several months, with involvement of respiratory muscles and 3 episodes of aspiration pneumonia. He died of respiratory failure at 11 months of age."
Patient 3 was a boy born in January 2006. Beginning at 2 months of age, he had chronic diarrhea, malabsorption, and failure to thrive. Recurrent episodes of pneumonia also developed, beginning when the boy was 4 months of age. OPV was administered at birth and at 2, 4, and 6 months of age. In October 2006, he was referred to hospital showing symptoms of acute paralysis of the left leg of 2 weeks’ duration, followed by involvement of his right leg and upper arms, accompanied by drowsiness, fever, and hypotonia. VDPV type 2 was isolated from both of his collected fecal specimens. The boy died <3 months after onset of paralysis after gram-negative sepsis in January 2007.” [10]
This study is out of Iran, published in 2010 and focuses on the increased susceptibility to VAPP of children who are immunodeficient. Of the 6 cases discussed, only one child survived. OPV is not recommended to children who are immunodeficient so it seems the immunodeficiency develops during or after receiving it in the rest of these cases.
It is noted that the OPV may shed for 8-12 weeks after administration. [11]
The CDC claims the inactivated polio vaccine (IPV) contributes to herd immunity but this is impossible to claim without studying it closely and proving that no shedding occurs if they were to be exposed with the actual infection. It can be studied no more closely than having an experiment where the vaccinated are actually challenged with the disease. The following study accomplished this by vaccinating first with IPV as infants and then challenging with the OPV which contains live virus and then measuring stool samples for shedding. There were 3 groups. Group A received a combination of DPT-Hib-IPV at 6, 10, and 14 weeks of age. Group B, the control group, received a combination of DPT vaccine and Hib vaccine at 6, 10, and 14 weeks. Group C received the DPT-Hib-IPV combination at 8 and 16 weeks.
All three groups were positive for vaccine strain polio viruses in their stool a week later. [12] This is conclusive proof that the IPV does not stop transmission in the event the vaccinated is later exposed to the virus.
SV40 Simian virus number 40, is a monkey virus that has been found in several types of cancers, including lung mesotheliomas, several types of brain tumors, and bone, breast, colon, and kidney tumors. [13] Vaccines manufactured using monkey kidney on which to grow their viruses up into the 1980s have been definitively noted [14] to contain a carcinogenic monkey virus that can result in cancer in a portion of the millions who were given them. [15]
“How a virus dubbed “the perfect war machine” by Dr Carbone because it affects at least four major cellular mechanisms that either promote cancer or interfere with cancer-fighting defensese, could be impacting countries that continue using oral polio vaccines by the ton today, is anyone’s guess. How much of the abrupt rise in human cancer rates since the introduction of monkey products into the human population is due to SV40 will also remain uncertain due to lack of precise research.” –Dissolving Illusions
Monkeys are still used in polio vaccine production today. According to Stanley Kops’ allegations, SV40 was and still is a potential risk in both the OPV and the inactivated polio vaccines. The only vaccine that doesn’t use monkey kidney cells, uses aborted fetal tissue instead. [16, 17]
Interesting side note, both of the rotavirus vaccines today are contaminated with two different pig viruses called “porcine circovirus (PCV) 1 and 2. …not known to cause disease in humans”. [18]
Very reassuring, since there’s no way to know if they cause disease or not until after it’s too late, unless they were to study this type of thing. In my experience, studies are not done until there is a cause for concern and even then will often be ignored for decades. The general attitude seems to point to the fact that they would just rather not know. They hope for the best and let people take that risk.
Fast forward to present day. No wild polio virus in the Western Hemisphere, barely any in the entire world, in fact. In 2017, there were only two cases of wild polio virus causing paralysis. Most years, its well under 10. But is that good enough? Not even close. It must be stomped out at all cost. And I do mean at all cost. So we march on despite any casualties. All worth it, I’m sure. This is war, after all. A few casualties are worth it in the name of eradicating a disease.
What about paralysis? How is that going? Now that we have new diagnoses for things that would have fallen under the polio criteria, does anyone know the numbers? What about children on ventilators? Some people celebrate the fact that we no longer have people in iron lungs but the threat of returning people to iron lungs is almost as ludicrous as threatening the “return” of polio.
In India, as the WHO tracks polio during the vaccination campaigns, reports of paralytic cases associated with wild-type poliovirus have declined but AFP (acute flaccid paralysis) has increased annually, reaching 60,000 new cases in 2011.
It just so happens that DDT is still used in India. And the cause of AFP is, of course, a huge mystery to everyone.
The OPV was discontinued here in the US in 2000 because of the contamination of vaccine strain polio in our water sources along with multiple complaints of paralysis after the vaccine but it continues to be used aggressively around the world, some reports say an Indian child is vaccinated with live vaccine 15 times by the time they are 5. [19]
Only 2 of the last 30 outbreaks of polio tracked by the WHO have been caused by wild polio over the last decade, tracking back to 2014. [20] This is undisputed fact. Once there is an admission that more damage is being caused than you are preventing, isn’t it time to just back off and leave nature alone? But no. The battle continues.
It has been reported in the Lancet that the incidence of AFP, especially non-polio AFP, increased drastically in India after an experimental, high-potency polio vaccine was introduced. [21] Worse still is that children identified with non-polio AFP are at more than twice the risk of dying than those with wild polio infection. [22] Shouldn’t eliminating paralysis be more important than simply replacing wild virus with vaccine virus regardless of the outcome? It seems that paralysis itself is nothing when compared to the horror contained in the tunnel vision of disease eradication.
Is it possible to eradicate something with a vaccine that causes the disease it’s trying to get rid of?
What about paralysis here in the US?
Dr Douglas Kerr from John Hopkins states, “Infants as young as five months old can get transverse myelitis, and some are left permanently paralyzed and dependent upon a ventilator to breathe…my colleagues at the Johns Hopkins Hospital and I hear about or treat hundreds of new cases every year.” [23] Does the public have any idea that there are hundreds of cases of something that is now called transverse myelitis that would historically have been called polio and is now leaving children permanently dependent on a modern version of the iron lung?
Approximately 33,000 people are afflicted by transverse myelitis in the Unites States, with 1,400 new cases per year. [24] There are up to 6,000 new cases of Gillian barre Syndrome (GBS) annually [25], and 75,000 new cases annually of aseptic meningitis.[26] All of these would previously have been diagnosed as polio. That’s a total of a possible 82,400 cases of polio like illness diagnosed every year. In contrast, polio pre vaccine was causing 17,000 cases in 1931 and at its peak in 1952, 21,000 cases of paralysis attributed to polio were diagnosed. [27]
This is why people ask the question, “Is polio really gone? Or has it just been renamed?”
What causes transverse myelitis? No one seems to have identified a sole cause, but the National Institute of Health lists vaccines as one of the causes, [28] and it’s listed in the adverse events or postmarketing surveillance sections of almost every vaccine package insert. It’s listed on page 5 of the varicella insert, for example. [29] I bet people would be shocked to find that an unlucky few are making the choice between their child being permanently paralyzed and a few days of chicken pox.
This article from last year was quite eerie to read after all the case reports I’ve been reading lately. Quotes like the following had a shiver going up my spine at the connections being so completely missed.
“Government health officials are urging U.S. doctors to be on the lookout for a mysterious condition that has begun to re-emerge in late summer and early fall, targeting healthy young children and leaving them paralyzed. There have been about 570 cases to date.
“There is no known way to prevent or treat it, and doctors are still unable to predict which children will develop paralysis. For those who do, the paralysis and weakness can last for months and it’s unclear how many will fully recover.
“We want clinicians and parents to be ready for a possible significant outbreak this year,” Anne Schuchat, principal deputy director of the CDC, said on a conference call with reporters. “One of the reasons we are reporting these results right now in July is because it’s every August that we start to see the uptick.
“Identifying potential patients early in the course of the disease is critical since the condition can quickly spiral from limb weakness to full-blown respiratory failure that requires intensive medical care, according to the CDC. Children who get the condition tend to be about 5 years old.”
What happens in August to five-year-olds? Hmmm. But I’m sure before-school-shots have absolutely nothing to do with this. It’s definitely just a coincidence.
Richard Carter, Breakthrough: The Saga of Jonas Salk, Trident Press, New York, 1955, p. 282
https://journals.sagepub.com/doi/abs/10.1191/1740774504cn011xx
https://academic.oup.com/aje/article-abstract/142/2/109/116270?redirectedFrom=fulltext (pp 14 and 16)
Paul Offit, MD, The Cutter Incident, Yale University Press, 2005, p. 89
https://jamanetwork.com/journals/jama/article-abstract/301686
A. Langmuir and N. Nathanson, “The Wyeth Problem”, September 6, 1955
https://www.cdc.gov/vaccines/schedules/hcp/imz/child-adolescent.html
https://www.fda.gov/files/vaccines%2C%20blood%20%26%20biologics/published/Package-Insert-IPOL.pdf
https://www.cdc.gov/vaccines/pubs/pinkbook/downloads/appendices/b/excipient-table-2.pdf
https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(07)60531-5/fulltext
https://www.goodreads.com/book/show/1635281.The_Autoimmune_Epidemic
https://www.chop.edu/conditions-diseases/transverse-myelitis
https://www.merck.com/product/usa/pi_circulars/v/varivax/varivax_pi.pdf